Outgoing CDC Director Dr. Robert Redfield, MD is a FAILURE as a leader.

Dr. Robert R. Redfield, MD – 18th Director of the Centers for Disease Control and Prevention

But allow me to be more explicit.

Dr. Robert R. Redfield, MD is a goddamn moron for whom I have no professional respect.

Why do I write that?

Like his soon-to-be former boss – the outgoing 45th President – he is, and remains, an ineffectual (mis)leader, under whose oversight the agency, like America, has languished, and suffered significant loss. Furthermore, also like his soon-to-be former boss – the outgoing 45th President – he refuses to accept responsibility for any of it.

The outgoing President, himself an utterly incompetent goddamn know-it-all moron, has a knack and penchant for identifying and naming the most utterly incompetent boobs to important positions within the administration.

Does that mean Redfield is a “most utterly incompetent boob”?

Not necessarily.

Of course, if you’ve been paying the least bit of attention for the past year, or so, you’d know the moronic and utterly idiotic things he’s done, and the equally moronic and contradictory things he’s said to justify, in response to the coronavirus pandemic in the United States.

But here below, read what he said recently in an interview with NPR’s Mary Louise Kelly on All Things Considered. • [Commentary in italics, within brackets, preceded with a •, following Redfield’s remarks.]

Outgoing CDC Director Warns Of Pandemic’s Peak:
“We’re About To Be In The Worst Of It”

January 15, 20216:07 PM ET
Heard on All Things Considered
by Mary Louise Kelly

Mary Louise Kelly: “Why has the U.S. done so much worse than the rest of the world?”

Robert Redfield: “I think this virus has a unique ability to have differential pathogenesis in different people. And what it really does is it exploits the underlying health condition of the individual it infects. And so, I would argue one of the reasons we’re having more significant death in this country than, say, Sweden is because unfortunately, the underlying health conditions — with obesity, diabetes, heart disease, kidney disease and the significant health disparities that we have in these illnesses in our nation — haven’t been effectively addressed.”
• [Pass the buck, please. There is EXACTLY ONE research paper in the entire National Library of Medicine with the subject “differential pathogenesis” in its title which is about COVID-19: Molecular Aspects of COVID-19 Differential Pathogenesis. The gist of the paper’s findings is that a type of the female hormone estradiol increases the levels of Angiotensin-Converting Enzyme 2 (ACE2), and that ACE2 apparently has some role in preventing severity of symptoms associated with COVID-19 infection, although, “ACE2 expression is dramatically reduced with aging in both genders. The levels of ACE2 expression, which could be sex- and age-dependent, have a protective role against lung and kidney injuries that could impact the severity of COVID-19 illness in male vs. females and old vs. young individuals.” As well, TMPRSS2, a cellular transmembrane protease, has a role in the severity of the disease, insofar as the “expression levels of TMPRSS2 protein are regulated by levels of androgen and androgen receptors … women and children have a lower level of androgen and androgen receptors than men, and therefore, TMPRSS2 could play a potential role in the severity of COVID-19 pathogenesis in men.” The study’s authors also write that, “it could be possible that the expression levels of ACE2 and TMPRSS2 impact virus infectivity and pathogenesis among different groups of individuals, considering the variation in the expression levels in older men compared to the women and children.” It is well known that individuals with comorbidities of hypertension, diabetes mellitus, heart diseases, and cerebrovascular disease, are at increased risk for poor outcomes, and increased risk of mortality, if infected with COVID-19, and the authors specifically state that the treatments for such conditions may very well place such individuals at increased risk for poor outcomes by writing that they “could be linked to the ACE2 function during SARS-CoV-2 infection and the cardio-metabolic treatments that may interfere withACE2–virus interaction.” The study’s authors conclude that, “variations in the expression levels of SARS-CoV-2 receptors and co-receptors, due to physiological and co-morbidity conditions, could impact the differential pathogenesis of COVID-19.” Contrary to what Dr. Redfield says, the virus does NOT have “a unique ability to have differential pathogenesis in different people.”]

Kelly: “But in terms of how the U.S. has responded, in terms of how the CDC has responded … are you able to defend the Trump administration’s record on this as anything other than a catastrophic failure?”

Redfield: “Well, I’m actually very proud of the response that CDC has done. I think if I have one criticism that